| 폐쇄수면무호흡과 뇌졸중; 폐쇄수면무호흡은 뇌졸중의 원인인가 결과인가? |
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김요식, 박현영 |
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원광대학교의과대학 신경과학교실, 임상의학연구소 |
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| Obstructive Sleep Apnea and Stroke; Cause or Consequence? |
| Yoski Kim, Hyun-Young Park |
| Department of Neurology and Clinical Research Center, Wonkwang University, Iksan, Korea |
| Corresponding Author:
Yoski Kim ,Tel: +82-63-859-1412, Fax: +82-63-842-7379, Email: yosik@wonkwang.ac.kr |
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Received: June 5, 2008 Accepted: June 25, 2008 Published online: June 30, 2008 |
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| ABSTRACT |
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Obstructive sleep apnea (OSA) is a common disorder which noted in 2-4% of general population. Patients considered it as a
non-significant event and do not try to evaluate and treat it. The common manifestation of OSA is related to sleep fragmentation and
is an excessive daytime sleepiness, decreased concentration, memory impairment and decreased job activity. Furthermore, OSA has
negative effects on cardiovascular system. It increases prevalence and incidence of stroke and ischemic heart disease. OSA is associated
with early neurological worsening and delay recovery from neurological deficit after stroke. OSA has negative effects on hypertension,
platelet activation, fibrinogen, cardiac arrhythmia, endothelial function and IMT (intima-media thickness) in carotid artery. Treatment
with continuous positive airway pressure (CPAP) has demonstrated reduction in risk factor abnormality and hope to decrease risk of
cardiovascular events including stroke. However, OSA has not been considered as an independent risk factor for stroke, it needs conclusive
evidence from well designed epidemiological studies. |
| Keywords:
Obstructive sleep apnea | Stroke | Positive airway pressure |
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